Showing posts with label #NURSING NOTES. Show all posts
Showing posts with label #NURSING NOTES. Show all posts

ONSET OF LABOR IN ENGLISH

                                             

                                  ONSET OF LABOR IN ENGLISH

               watch my youtube video to understand this topic in easy way-

    https://www.youtube.com/watch?v=nzEoW-_jD5o

ONSET OF LABOR-

 

Labor-

Series of events that take place in the genital organs in an effort to expel the viable products of  conception (fetus, placenta and the membranes) out of the womb through the vagina into the outer world is called Labor.

Labor (EUTOCIA) is called normal following criteria are fulfilled-

(1) Spontaneous in onset and at term.

(2) With vertex presentation.

(3) Without undue prolongation.

(4) Natural termination with minimal aids.

(5) Without having any complications affecting the       health of the mother and/or the baby.

 

Cause of onset of labor

There is no clarity about what causes onset of labor still some theories are-

Uterine distension: Stretching effect on the myometrium by the growing fetus and liquor amnii can explain the onset of labor at least in twins or poly-hydramnios.

Uterine stretch increases gap junction proteins, receptors for oxytocin and specific contraction associated proteins (CAPs).

Feto-placental contribution: fetal hypothalamic-pituitary- adrenal axis-  Just before onset of labor there is increased CRH  in fetal hypothalamus→ increased release of ACTH from fetal pituitary and→ increased cortisol secretion from fetal adrenals → accelerated. This event leads to production of estrogen and prostaglandins from the placenta

This estrogen — Increases the release of oxytocin from maternal pituitary. — Promotes the synthesis of myometrial receptors for oxytocin, prostaglandins and increase in gap junctions in myometrial cells.

— Accelerates lysosomal disintegration in the decidual and amnion cells resulting in increased prostaglandin (PGF2α) synthesis. — Stimulates the synthesis of myometrial contractile protein—actomyosin 

  and  Increases the excitability of the myometrial cell membranes.

Progesterone:   Progesterone levels  fall before labor (due to fetal cortisol and de-hydro-epi-androsterone sulfate (DHEA-S) . Change in the estrogen : progesterone ratio rather is linked with prostaglandin synthesis.

Prostaglandins: Prostaglandins are the important factors, which initiate and maintain labor.

 

Pre Labor Stage-

PRELABOR: (premonitory stage): The premonitory stage may begin 2–3 weeks before the onset of true labor in primigravidae and a few days before in multiparae. The features are inconsistent

and may consist of the following:

        Lightening

        Cervical changes

        Appearance of false labor pain

 

Lightening-

A few weeks prior to the onset of labor especially in primigravidae, the presenting part sinks into the true pelvis. It is due to active pulling up of the lower pole of the uterus around the presenting part. 

This diminishes the fundal height and hence minimizes the pressure on the diaphragm. It is a  welcome sign as it rules out cephalopelvic disproportion and other conditions preventing the head from entering the pelvic inlet.

Cervical changes-

Cervical changes: A few days prior to the onset of labor, cervix becomes ripe. A ripe cervix is

(a)    soft,

(b)    80% effaced (<1.5 cm in length),

(c) Admits one finger easily, and

(d) Cervical canal is dilatable.

Appearance of False labor pain-

False labor pain is –

      (i)          Dull in nature,

(ii) confined to lower abdomen and groin,

(iii) not associated with hardening of the uterus,

(iv) they have no other features of true labor  pain 

(v) usually relieved by enema or sedative.

 

Identifying the onset of labor-

The onset of labor is a process and is very difficult to identify exactly when the painless (sometimes painful) contractions of pre-labour develop into the progressive rhythmic contractions of actual labour. Onset of labor is assumed to start when rhythmic pattern of contraction starts and results in effacement and dilatation

True labor pain is : (i) Painful uterine contractions at regular intervals, (ii) frequency of contractions increase gradually, (iii) intensity and duration of contractions increase progressively, (iv) associated with “show”, (v) progressive effacement and  dilatation of the cervix, (vi) descent of the presenting part, (vii) formation of the “bag of forewaters” and (viii) not relieved by enema or sedatives.

Show: With the onset of labor, there is profuse cervical secretion. Simultaneously, there is slight oozing of blood from rupture of capillary vessels of the cervix and from the raw decidual surface caused by separation of the membranes due to stretching of the lower uterine segment. Expulsion of cervical mucus plug mixed with blood is called “show”.

Formation of bag of waters- Due to stretching of the lower uterine segment and With the dilatation of the cervical canal, the lower pole of the fetal membranes becomes unsupported and tends to bulge into the cervical canal. As it contains liquor, which has passed below the presenting part, it is called “bag of waters”.  


ECLAMPSITA IN ENGLISH

                                                 

                                          ECLAMPSITA  IN ENGLISH

               watch my youtube video to understand this topic in easy way-

  https://www.youtube.com/watch?v=2MWlhm8BepE

ECLAMPSIA-

Eclampsia is a neurological condition associated with preeclampsia, manifesting with tonic-clonic convulsions in pregnancy that are not ssocited with other conditions such as epilepsy

Eclampsia can develop any time from 20 weeks’ gestation up to 6 weeks postpartum. It is an medical emergency and should be dealt with a team of physician and gynecologist

 

CLINICAL MANIFESTATIONS-

        Preceded by symptoms of severe preeclampsia:

        Severe headache

        Visual disturbances

        Epigastric/RUQ pain

        Nausea, vomiting

        Oliguria

        Seizures

        Seizure chacacteristics

        Tonic–clonic convulsions

        Usually self-limiting (1–2 minutes)

        It May progress to coma

        Seizure  may recur

         

COMPLICATIONS-

        Complications (Maternal)

        Cerebral hemorrhage

        Pulmonary edema

        HELLP syndrome

        Renal failure

        Disseminated Intravascular Coagulation (DIC)

        Death

        Complications (fetal/neonatal)

        Intrauterine Growth Restriction (IUGR)

        Preterm birth

        Hypoxia/asphyxia

        Stillbirth

        Neonatal death

 

CAUSES AND RISK FACOTRS

Pre-eclampsia  is the precursor of eclampsia so all risk factors of preeclampsia are also risk factors for ecclampsia

 

DIAGNOSTIC INVESTIGATIONS-

• Urine examination for protein

• Blood pressures assesment

• Complete blood count

• Serum electrolytes

• Liver function test

• Serum Urea and Creatinine

• Ultrasound

• Doppler velocimetry

• Electro Encephalo Graphy   (EEG)

 

MANAGEMENT-

Immediate Management-

        Assessment and maintenance of Airway, Breathing, Circulation (ABC)

        Place woman in left lateral position

        Maintain airway, suction if needed

        Oxygen administration

        Control seizures with Magnesium Sulphate

Magnesium Sulphate Therapy

     It is the Drug of choice for ecclampsia

        Loading: 4 g IV slowly + 10 g IM (5 g each buttock)

        Maintenance: 5 g IM every 4 hours

        Monitor for toxicity (respiratory depression, reflexes, urine output)

        Ante hypertensive drugs

Obstetric management

        Definitive treatment = Delivery

        Stabilize mother first

        Assess gestational age, fetal condition

        Induce labor or perform cesarean section if required

If the woman gives birth vaginally, syntometrine and ergometrine should be avoided to manage the third stage of labour and oxytocin

used instead.

NURSING MANAGEMENT-

        Management during seizures

        Stay with patient and call for help

        Provide Position in left lateral side

        Loosen all tight clothing

        Maintain airway, suction secretions

        Protect from injury (do not force objects into mouth)

        Management after seizures

        Monitor vital signs, fetal heart rate

        Insert IV line for fluids/medications

        Administer MgSO₄ & antihypertensives as prescribed

        Monitor urine output (≥30 ml/hr)

        Document events carefully

        The baby is likely to be initially cared for on the neonatal unit and the woman should be taken to see her babyas soon as her condition permits.

        Breastfeeding is to be encouraged and psychological support given by themidwife/nurse and neonatal staff.


PRE ECLAMPSITA IN ENGLISH

                                                       

                                             PRE ECLAMPSITA  IN ENGLISH

               watch my youtube video to understand this topic in easy way-

 https://www.youtube.com/watch?v=C38al47A-AI

PRE ECLAMPSIA-

Hypertensive disorders of pregnancy-

q  Gestational hypertension

q  Pre Eclampsia

q  Chronic hypertension

q  PreEclampsia superimposed on chronic hypertension

q  Eclampsia

Pre-eclampsia is an idiopathic  condition of pregnancy characterized by proteinuria and hypertension (>140/90 mmHg) presenting after 20 weeks of pregnancy in a woman who

previously had normal blood pressure.

Preeclampsia is defined as new onset of hypertension (≥140/90 mmHg) and proteinuria which occurs after 20 weeks of gestation in previously normotensive woman

 

CLINICAL MANIFESTATIONS-

• blood pressure: systolic >140 mmHg or diastolic >90 mmHg 

• proteinuria 

• edema – may be detectable on examination.

-Ankle edema initially and than more

 generalized edema that pits on pressure .

-Edema is seen on pre-tibial surface, face, hands, abdomen and sacrum.

 

CAUSES AND RISK FACTORS

Pre-eclampsia is an idiopathic  condition Risk factors includes-

• Maternal age (<20 and >40 years)

• Family history of pre-eclampsia

• Pre-eclampsia in a previous pregnancy

• Pregnancy after assisted reproductive technology

• Obesity

• Pre-existing diabetes mellitus type 1

 • Pre-existing hypertensive disease

• Pre-existing medical conditions, e.g. renal disease,

systemic lupus erythematosus (SLE), rheumatoid

Arthritis

• Developing a medical disorder during pregnancy,

e.g. venous thromboembolic disease (VTE), such as

antiphospholipid (Hughes) syndrome (APS),

gestational diabetes, gestational hypertension

• First pregnancy

• Multiple pregnancy

• Developing infection with inflammatory response

• Hydropic degeneration of the placenta

 

DIAGNOSTIC INVESTIGATIONS-

• Urine examination for protein

-Urine sample or 24 hour urine collection to quantify the proteinuria (>300 mg) and determine the ratio of protein to creatinine (>30 mg/mmol).

• Complete blood count

• Serum electrolytes

• Liver function test

• Serum Urea and Creatinine

• Ultrasound

• Doppler velocimetry

 

MANAGEMENT-

• Anti-hypertensives such as methyldopa

and nifedipine.

•In severe case hospital admission may be required with IV antihypertensive drugs.

• Magnesium sulphate prophylaxis may be added.

• expedite the birth of the baby and placenta.

• Induction of labour will be determined by the

obstetrician, and is likely to be at 37 weeks for mild preeclampsia 34–36 weeks for moderate pre-eclampsia and at 34 weeks for severe hypertension.

• Birth should be earlier in the event of uncontrolled blood pressure or fetal or antenatal complications, with caesarean section.

 

NURSING MANAGEMENT-

        Nursing assesment including detailed history taking

        Frequent BP monitoring as per order

        Assessment of Urine output & reflexes

        Fetal heart rate monitoring to assess fetal distress

        Should administer prescribed drugs on time

        Maintain fluid balance oral or IV as per order

        Observe for seizures 

        Provide psychological support

Provide health education to patient and family  about-

        disease & warning signs

        Diet: low salt, adequate protein and

        Importance of regular ANC check-ups


AYUSHMAN BHARAT PM JAY IN HINDI

                                                                                 AYUSHMAN BHARAT PM JAY IN HINDI                     watch m...